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This page provides a listing of abbreviations used regularly on Palaeos.org that may not be linked to an explanatory page at every occurrence:
Mya: million years ago
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Often times the breakdown or damage in the sequence affects the arteries, muscles, and surrounding tissues of the penis, and this breakdown is most commonly the result of a disease. Diseases that commonly cause ED include:
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Diseases (e.g., diabetes, kidney disease, alcoholism, atherosclerosis) account for as many as 70% of chronic ED cases and psychological factors (e.g., stress, anxiety, depression) may account for 10–20% of cases. Between 35 and 50% of men with diabetes experience ED.
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Physical inactivity, which can lead to obesity, increases the risk of erectile dysfunction.
In the past, the placement of prosthetic devices within the corpora was the only effective therapy for men with organic ED. Now, this is the last option considered, even though more than 90% of men with an implant would recommend the procedure to their friends and relatives.
Patients A total of 268 men (mean age, 57 years) with erectile dysfunction (mean duration, 5.6 years) and diabetes (mean duration, 12 years).
Erectile dysfunction is when an erection is consistently difficult or impossible to produce, despite arousal. There are various and often multiple underlying causes, some of which are treatable medical conditions. The most important organic causes are cardiovascular disease and diabetes, neurological problems (for example, trauma from prostatectomy surgery), hormonal insufficiencies and drug side effects.
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The appropriate homeopathic remedy can help move the body and mind toward healing. A trained homeopathic practitioner is needed to identify and prescribe a deep-acting, constitutional remedy.
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Methods and results 285 patients with CAD divided into three age-matched groups: group 1 (G1, n = 95), ACS and one-vessel disease (1-VD); group 2 (G2, n = 95), ACS and 2,3-VD; group 3 (G3, n = 95), chronic CS. Control group (C, n = 95) was composed of patients with suspected CAD who were found to have entirely normal coronary arteries by angiography. Gensini's score used to assess extent of CAD. ED as any value < 26 according to the International Index of Erectile Function (IIEF). ED prevalence was lower in G1 vs. G3 (22 vs. 65%, P < .0001) as a result of less atherosclerotic burden as expressed by Gensini's score [2 (0-6) vs. 40 (19-68), P = 0.0001]. Controls had ED rate values similar to G1 (24%). Group 2 ED rate, IIEF, and Gensini's scores were significantly different from G1 [55%, P < 0.0001; 24 (17-29), P = 0.0001; 21 (12.5-32), P < 0.0001] and similar to G3 suggesting that despite similar clinical presentation, ED in ACS differs according to the extent of CAD. No significant difference between groups was found in the number and type of conventional risk factors. Treatment with beta-blockers was more frequent in G3 vs. G1 and G2. In G3 patients who had ED, onset of sexual dysfunction occurred before CAD onset in 93%, with a mean time interval of 24 [12-36] months. In logistic regression analysis, age (OR=1.1; 95% confidence interval (CI), 1.05-1.16; P = < 0.0001), multi-vessel vs. single-vessel (OR=2.53; 95% CI, 1.43-4.51; P = 0.0002), and CCS vs. ACS (OR=2.32; 95% CI, 1.22-4.41; P = 0.01) were independent predictors of ED.
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