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Phyllocerida

Systematics

  • Class:
  • Subclass:
  • Superorder: Neoammonoidea
  • Order: Phyllocerida

The Phyllocerida, sometimes known as the Phylloceratida or as the suborder Phyllocerina, is a small but persitant stock of neoammonoids, characterized by phlloid saddle endings in the suture, that gave rise to all post Triassic ammonoids. Phylloceratids first appeared in the late Scythian (Lower Triassic) with the Ussuritidae, probably derived from the Dieneroceratidae.

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The Ussuritidae gave rise to the Discophyllitidae at the beginning of the Late Triassic (Carnian), but continued to the end of the period. The Discophyllitidae, which also continued well into the Raetian, give rise to the mostly Jurassic and Cretaceous Phylloceratidae, which forms the bulk of the order. The Ussuritidae or Discophyllitidae also gave rise to the Lytocerida which is the source of most of the Jurassic ammonitids

The Phylloceratidae may have given rise to the Juraphyllitidae, a small family confined to the Lower Jurassic, although the Juraphyllitidae may have evolved separately from the Discophyllitidae, and to certain of the Ammonitida. Otherwise the Phylloceratidae are the only phylloceratids of the post Triassic. The Phylloceratidae gave rise to the Psiloceratidae (Psilocerataceae, Ammonitida) near the beginning of the Jurassic and to the Desmoceratidae (Desmocerataceae) in the Lower Cretaceous (Valanginian) which is the root stock for the principal Cretaceous ammonitids


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The Ussuritidae are evolute with rounded venters and little or no ornamentation. Sutures have primitive, monophyllic saddle endings. The Ussuritidae are also known as the Monophyllitidae.

The Discophyllitidae are similar in form to the Ussuritidae but have diphyllic or triphllic endings to the principal saddles.

The Phylloceratidae are characterized by very thin, smooth, involute shells, usually without ribbing, and in many with fine growth lines. Sutures are complex with major and minor branches of saddles with double, tripple, and even quadruple phylloid and spatulate endings. Contains two subfamilies, the Phylloceratinae without flares and usually without constrictions, and the Calliphylloceratinae regularly with constrictions or flares, or both.

The Juraphyllitidae are compressed, evolute, with a modified body chamber that usually have coarse ventral ribbing. A few genera are more involute, but still compressed. The first lateral saddles are diphyllic, the rest diphyllic or triphyllic.

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  • Arkell et al, Mesozoic Ammonoidea. Treatise on Invertebrate Paleontology, Part L. R.C. Moore,(ed) 1957.


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On day 12 (CT26WT) or day 5 (C26GM), all mice received carboxyfluorescein diacetate succinimidyl ester (CFSE)–labeled splenocytes pulsed with the MHC class I–restricted AH-1 peptide (CFSEhigh), admixed with CFSE-labeled splenocytes pulsed with the irrelevant hemagglutinin (HA)-peptide (CFSElow). In vivo T cell cytotoxicity was determined 40 h later (Fig. 2). These time points were chosen based on the kinetics of tumor outgrowth observed in Fig. 1 A and Fig. 1 B, respectively, when tumor size significantly differed between untreated and sildenafil-treated mice. As expected, an endogenous AH1-specific immune response was observed in the vaccinated mice as compared with their tumor-bearing counterparts. PDE5 inhibition in the vaccine-primed mice failed to augment antigen-specific CD8 responsiveness compared with no treatment. In contrast, tumor-bearing mice treated with sildenafil early after tumor challenge generated antigen-specific immunity that was significantly greater then that observed in their untreated counterparts and similar, or even superior, to that induced by vaccination. Collectively, this is the first indication that PDE5 inhibitors can modulate antitumor immunity. Because the sildenafil-mediated antitumor immune response does not completely eradicate tumors, tumor escape mechanisms may be associated with their outgrowth. To test this hypothesis, the parental CT26WT cell line, as well as the CT26 tumor removed on day 24 from sildenafil-treated mice (either AH-1 pulsed or unpulsed), and BALB/c splenocytes were incubated with either AH-1 peptide-primed (Fig. S1 C) or tumor-primed (Fig. S1 D) effector T cells. Although effector T cells recognized the parental CT26WT line and released IFN- in the assay, they failed to recognize the sildenafil-derived tumor. Its recognition, however, was restored by loading the sildenafil-derived tumor with the AH-1 peptide. (Fig. S1, C and D). These results suggest that the immune response in sildenafil-treated mice does not result in complete tumor eradication but rather in the selection of antigen-escape variants. How is erectile dysfunction diagnosed? Buy Online Removing tension ring within 30 minutes recommended - Headaches and flushing ( may sham 1 in 10 people or more); All the patients then completed questionnaires regarding treatment side effects six months, 12 months, and 24 months following radiation. Last year, the FDA's scientific advisory board criticized the agency for ignoring more than 100 academic and government studies that linked BPA with health effects. The Obama administration has pledged a "fresh look" at the issue, and the FDA is expected to complete that review by the end of this month. 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